"When your children need to seriously calculate their chances of being murdered in a place of learning, you’re doing it wrong as a society.
The U.S. doesn’t have a children’s culture problem, or a too-many-doors problem, or a mental health problem.... that other countries don’t already already have.
The U.S. has a gun problem."
School shootings are so common that students talk in frighteningly practical terms about the location of doors and windows in their classrooms as risk factors. They calculate escape routes. And they ponder hiding spots in wide-open rooms.
Aggressively feminist, zombie worms that originated in the Cretaceous period will feast on the bones of animals by dissolving them with acid juice.
Should've been Sarlacc.
But it was a really fun article to read.
Chlorofluorocarbons are the main class of chemical that depleted the ozone layer in the stratosphere. Measurements reported in Nature reveal that emissions of t...hese compounds are rising again, despite international rules restricting their use. This News & Views article discusses the findings.
It seems like every other day there’s a new headline telling you what’s giving you cancer or what’s preventing cancer (I’ve found articles saying coffee does bo...th). But how much is in your control? What’s been proven to lower your risk? What hasn’t? And how much do you have to worry about every headline that says “life is probably killing you”?
We took a dive into the data for my latest piece at SELF to see what you should do (and shouldn’t bother with) to limit your risk for cancer. Hint: sunscreen.
Fluoride: your teeth need it.
Here's 4 reasons why our teeth need it (from the American Dental Association):
1. Prevents tooth decay
2. Protects against caviti...es
3. Shown to be safe and effective
4. Saves money
Benefits of fluoride: https://www.nhmrc.gov.au/…/20160824_evidence_report_final_1…
"It is a true fact that the science of the microbiome is an emerging field, with what we don’t know looming larger than what we do know. It’s a field where we s...hould approach this issues with real humility regarding what we don’t know and what we might find out. There are those however that have seized on the uncertainty of microbiome research to insist that implausible ideas should be taken seriously regardless of a lack of evidence or credible mechanisms. Likewise, those people will use the uncertainty of microbiome science to dismiss strong evidence and analysis with some hand waving and chin stroking about “We just don’t know.”
This is irresponsible and dishonest. In this case, we have a lot epidemiogical evidence on the health effects of glyphosate from the Agricultural Health Study. And we have a lot of observational animal feeding trials. And it’s pretty clear that gut flora have access to plenty of amino acids without having to synthesize them. Insisting that there is some health effect that is still eluding our detection with some invocation of what we don’t yet know about the microbiome is not rigorous skepticism, it’s granting yourself a license to believe whatever you want despite the evidence or lack there of. It’s all too often used as a Get Out of Jail Free Card when the thing you want to believe isn’t supported by the evidence. That’s not science, that’s a cheap debate tactic."
In JCI Insight: Inflammatory monocytes linked to sepsis-associated cognitive dysfunction
Sepsis is a life-threatening condition that acc...ounts for substantial morbidity and mortality in hospitalized patients. A majority of those that survive will exhibit long-term cognitive dysfunction, the etiology of which is unknown and for which treatment options are limited. Graciela Andonegui, Erin Zelinski and colleagues evaluated cognitive function in patients recovered from sepsis-associated delirium approximately one year after hospital discharge. Compared to control subjects, those who had sepsis-associated delirium scored lower on spatial and pattern recognition memory-associated tests. In mice, pneumonia-induced sepsis recapitulated human phenotypes, including multi-organ dysfunction, encephalopathy, and spatial memory defects. In both humans and mice, sepsis promoted increased expression of myeloid cell-recruiting chemokines, with murine models showing a notable increase in neutrophil and inflammatory CCR2+ monocytes (see accompanying images), and microglia activation. Importantly, prevention of inflammatory monocyte recruitment in septic mice markedly reduced neuroinflammation and prevented cognitive impairment, suggesting CCR2+ monocytes be further explored as a therapeutic target.